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Toxicological Sciences 2007 98(1):1-4; doi:10.1093/toxsci/kfm111
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© The Author 2007. Published by Oxford University Press on behalf of the Society of Toxicology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Arsenic: Extension of its Endocrine Disruption Potential to Interference with Estrogen Receptor-Mediated Signaling

Walter H. Watson1 and James D. Yager

Division of Toxicological Sciences, Department of Environmental Health Sciences, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland 21205

1 For correspondence via e-mail: wwatson@jhsph.edu.

Received April 30, 2007; accepted May 2, 2007

The first 150 words of the full text of this article appear below.

Human exposure to arsenic (As) has become an important public health concern. Exposures most commonly occur through consumption of drinking water where the current maximum contaminant level of As, set by the U.S. Environmental Health Administration and the World Health Organization, is 10 µg/l. However, contamination from natural sources in various areas of the United States such as New England and the Southwest, and regions of the world, such as Bangladesh and Taiwan can reach hundreds of micrograms/liter in wells. Exposures can also occur in occupational settings where arsenic is used to manufacture pesticides and wood preservatives, and superfund sites where industrial waste was disposed. An example of the issue and concern is represented by this recent headline which appeared in the Baltimore Sun on Friday, 20 April 2007. "ARSENIC FORCES CLOSING OF PARK—Tests show high levels in soil; city locks gates of Swann Park." Swann Park is located next . . . [Full Text of this Article]


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