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Toxicological Sciences 74, 228-229 (2003)
Copyright © 2003 by the Society of Toxicology

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Urmila P. Kodavanti1, Allen D. Ledbetter1, Mette C. Schladweiler1, Daniel L. Costa1, Carolyn F. Moyer2, Russ Hauser3, David C. Christiani3 and Abraham Nyska4

1 Pulmonary Toxicology Branch, Experimental Toxicology Division, National Health and Environmental Effects Research Laboratory, Office of Research and Development, U.S. Environmental Protection Agency, Research Triangle Park, NC 27711 2 Pathology Associates, Inc., Raleigh, NC 27606 3 Harvard School of Public Health, Boston, MA 02115 4 Laboratory of Experimental Pathology, National Institute of Environmental Health Sciences, Research Triangle Park, NC 27709

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To the Editor:

The Letter to the Editor regarding our recent article, "Inhaled Environmental Combustion Particles Cause Myocardial Injury in the Wistar Kyoto Rat," suggests a potential mechanism by which cardiac pathology may occur after long-term inhalation of particulate matter containing bioavailable zinc. Our paper is the first to demonstrate pathological myocardial lesions in Wistar-Kyoto rats following inhalation exposure to combustion emission particles rich in bioavailable zinc (Kodavanti et al.. . . [Full Text of this Article]


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