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Toxicological Sciences 2004 81(1):254-255; doi:10.1093/toxsci/kfh186
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Toxicological Sciences vol. 81 no. 1 © Society of Toxicology 2004; all rights reserved.

Letter to the Editor

Janine Ezendam1,2,3, Joseph G. Vos2,3 and Raymond Pieters1

1 Institute for Risk Assessment Sciences, Utrecht University,P.O. Box 80.176, 3508 TD, Utrecht, The Netherlands 2 National Institute for Public Health and the Environment, P.O. Box 1, 3720 BA, Bilthoven, The Netherlands 3 Faculty of Veterinary Medicine, Department of Pathobiology, Utrecht University, P.O. Box 80.150, 3508 TD, Utrecht, The Netherlands

The first 10% of the full text of this article appears below.

To the Editor:

We have read the comments of Dr. Nanne Bloksma on our paper, "Hexachlorobenzene-induced immunopathology in Brown Norway rats is partly mediated by T cells" (Ezendam et al., 2004aGo).

We appreciate her comments and would like to respond to them. The study mentioned by Bloksma indeed showed that in genetically athymic WAG/Rij rats and in thymectomized Brown Norway (BN) rats HCB-induced immune effects still occurred. Skin lesions in the athymic rats could not be assessed properly because of preexisting inflammatory skin lesions . . . [Full Text of this Article]


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